HIGHLIGHT
重要成果
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- Early Growth Response 1 is an Early Signal Inducing Cav3.2 T-type Calcium Channels During Cardiac Hypertrophy.
- Cardiovasc Res., Aug 08, 2013
- The Cav3.2 T-channel plays a pivotal role in inducing calcineurin/nuclear factor of activated T cell (NFAT) signaling during cardiac hypertr ......
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- Quantitative phosphoproteomic study of pressure-overloaded mouse heart reveals dynamin-related protein 1 as a modulator of cardiac hypertrophy.
- Mol Cell Proteomics., Jul 23, 2013
- Pressure-overload stress to the heart causes pathological cardiac hypertrophy, which increases the risk of cardiac morbidity and mortality. ......
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- The Cav3.1 T-type calcium channel is required for neointimal formation in response to vascular injury in mice.
- Cardiovasc Res., Aug 10, 2012
- Restenosis is an undesirable consequence following percutaneous vascular interventions. However, the current strategy for preventing resteno ......
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- Cav3.2 T-Type Ca2+ Channel-Dependent Activation of ERK in Paraventricular Thalamus Modulates Acid-Induced Chronic Muscle Pain.
- J Neurosci., Aug 04, 2010
- Treatments for chronic musculoskeletal pain, such as lower back pain, fibromyalgia, and myofascial pain syndrome, remain inadequate because ......
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- The CaV3.2 T-Type Ca2+ Channel Is Required for Pressure Overload–Induced Cardiac Hypertrophy in Mice.
- Circulation Research., Jan 02, 2009
- Voltage-gated T-type Ca2+ channels (T-channels) are normally expressed during embryonic development in ventricular myocytes but are undet ......
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