1992, PhD, University of California, Los Angeles (UCLA), USA
2020 ~ date, CEO, Emerging Infectious Disease Division (EIDD), Biomedical Translation Research Center (BioTReC), Academia Sinica
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Infection with Japanese encephalitis virus (JEV) can induce the expression of pro-inflammatory cytokines and cause acute encephalitis in humans. ?-oxidation breaks down fatty acids for ATP production in mitochondria, and impaired ?-oxidation can induce pro-inflammatory cytokine expression. To address the role of fatty-acid ?-oxidation in JEV infection, we measured the oxygen consumption rate of mock- and JEV-infected cells cultured with or without